[Abstract] [Full Text PDF] (in Japanese / 11631KB) [Members Only And Two Factor Auth.]

J.Jpn. Surg. Soc.. 56(7): 867-883, 1955


A STUDY ON THE STELLATE GANGLION BALOCK
PART 3. CLINICAL EXPERIMENTAL STUDY

Department of Neurosurgery, Niigata University School of Medicine (Director: Prof. Mizuho NAKATA)

Hiroshi FUKAI

Previously, from the results of the clinical experiences and animal experiments the author reported the mechanism of stellate ganglion blocks seemed to diminish cerebral vascular tone, dilate cerebrl vessel and improve cerebrfal circulation. Since then, clinical experiments were done to clarify the mechanism of this treatment and the following results were obtained.
1. There is no significant changes in the study of pulse, blood-pressure and EKG before and after block.
2. After the blocks so-called Horner's Syndrom is detected on the corresponding side, however the appeara ce of a sign of each component of it is not synchronous but various. Besides, no true enophthalmos is noticed.
3. Following the blocks a tendency of the increased skin temperature of face and finger on the corresponding side and of decreased skin temperature of those on the contralateral side, were observed.
4. An increase of the spinal fluid pressure occurred follwing the blocks.
5. Following the blocks a normalizing tendency of the abnormal EEG especially on the corresponding side, was recognized. But there are no parallel with improved EEG and clinical symptom.
6. The block leads to homolateral dilatation of retinaI vessels espcially arterioIes and it was observed in a number of cases in which clinical symptom improved.
7. After the blocks a tendency of decrease of cerbral vascular resistance and increase of cerebral blood flow, cerebral oxygen consumption and cerebral glucose consumption. were noticed.
8. The author confirmed from those findings and animal experiments reported previously that clinical favorable results following stellate block for cerebral circulatory disorders were derived from improved brain circulation caused by an changes in the intrinsic regulation of cerebral circulation by means of decreased cerebral vascular tone and dilated cerebral vessels.
(author's abstract)


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